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Integrative analysis of HIF binding and transactivation reveals its role in maintaining histone methylation homeostasis.

Abstract:
Adaptation to hypoxia is mediated through a coordinated transcriptional response driven largely by hypoxia-inducible factor 1 (HIF-1). We used ChIP-chip and gene expression profiling to identify direct targets of HIF-1 transactivation on a genome-wide scale. Several hundred direct HIF-1 targets were identified and, as expected, were highly enriched for proteins that facilitate metabolic adaptation to hypoxia. Surprisingly, there was also striking enrichment for the family of 2-oxoglutarate dioxygenases, including the jumonji-domain histone demethylases. We demonstrate that these histone demethylases are direct HIF targets, and their up-regulation helps maintain epigenetic homeostasis under hypoxic conditions. These results suggest that the coordinated increase in expression of several oxygen-dependent enzymes by HIF may help compensate for decreased levels of oxygen under conditions of cellular hypoxia.
Authors:
X Xia, ME Lemieux, W Li, JS Carroll, M Brown, XS Liu, AL Kung
Journal:
Proc Natl Acad Sci U S A
Citation info:
106(11):4260-4265
Publication date:
17th Mar 2009
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